Lumbar Radiculopathy assesment
- Fysiobasen
- Dec 19, 2025
- 5 min read
Lumbar radiculopathy is a neurological condition caused by compression or irritation of a lumbar nerve root, producing motor, sensory, and reflex changes in the corresponding myotomeĀ and dermatome. Although pain is notĀ part of the formal definition, patients commonly report radiating, sharp/electric pain, numbness, and paresthesia. The symptom experience is subjective and varies with a patientās interpretation and pain beliefs.¹

Radiculopathy vs. Radicular Pain:Ā they often coexist but can occur independentlyāradiculopathy may be present without pain, and radicular pain can occur without objective neurological signs.² Estimated prevalence is 3ā5%Ā of the population,³ ā“ and it is among the most common reasonsĀ for referral to spine surgery.āµ Among people with low back pain, 12ā40%Ā also show radiculopathy features.ā¶
Common Etiologies
The most frequent cause is disc herniationĀ with nerve root compression. Other causes include:
Degenerative changes:Ā spondylolisthesis, spinal stenosis, osteophytes
Trauma:Ā compression fractures
TumorsĀ within/adjacent to the canal
InfectionsĀ involving vertebrae or nerve roots
Vascular causes:Ā venous congestion, hemangioblastomas, AVMsā¶ āø
Red Flags ā Immediate Referral
Refer urgently if any of the following are presentā¹:
Fever without clear source
Unexplained weight loss
New urinary or fecal incontinence/retention
Saddle anesthesia
Gait ataxia
Prior or active malignancy
Body Charting & Symptom Mapping
Use a body chart and active exploration (palpation, provocation) beyond spontaneously reported areas to uncover latent or under-reported symptoms. This clarifies the radicular distribution and improves diagnostic precision.¹
Objective Examination ā Core Elements

Myotomes:Ā strength testing for segmental motor deficits
Reflexes:Ā patellar and Achilles; hyporeflexia may indicate root involvement
Sensation:Ā light touch/pinprick in dermatomal territories
Upper motor neuron screen:Ā Hoffmann, Babinski, ankle clonus to exclude central signs
Neurodynamic tests:Ā SLR, Slump (and Femoral nerve test for high lumbar roots)
Lumbar A/P ROM:Ā flexion, extension, lateral flexion/rotation (active & passive)
Repeated movement testing:Ā McKenzie/MDT principles as testāretest
Segmental palpation & accessory motion:Ā symptom reproduction, hypomobility/hyper-mobility
No single test is highly accurate alone. CombinationsĀ of historical features + clustered physical tests improve diagnostic value and guide management.¹ā°
Myotomes ā Lower Limb
Nerve Root | Primary Movement (Key Muscle Action) |
L2 | Hip flexion |
L3 | Knee extension |
L4 | Ankle dorsiflexion |
L5 | Great toe extension |
S1 | Ankle plantarflexion |
S2 | Knee flexion (hamstrings) |
How to test (manual muscle testing):Explain, position accurately, apply gradually increasing resistanceĀ for ~3 seconds (āhold, hold, holdā), grade 0ā5, compare bilaterally, and record patient-reported asymmetry even when objective strength seems equal.
Dermatomes ā Lower Limb
Dermatome | Typical Cutaneous Area |
L1 | Region around iliac crest and greater trochanter |
L2 | Anterior thigh (groin to mid-thigh/knee) |
L3 | Anterior thigh & knee; medial leg |
L4 | Lateral thigh, medial leg, dorsum of foot and hallux |
L5 | Postero-lateral thigh, lateral leg, dorsum of foot, medial sole, toes 1ā3 |
S1 | Posterior thigh & leg, lateral foot border |
Light touch testing tips:Ā patient supine/sitting, eyes closed; use fingertip/cotton; compare sides; use the same mapĀ consistently across patients.
Deep Tendon Reflexes
Reflex | Peripheral Nerve | Root Level | Strike Location | Expected Response |
Patellar | Femoral n. | L2āL4 | Patellar tendon (infrapatellar) | Knee extension |
Achilles | Tibial n. | S1āS2 | Achilles tendon | Ankle plantarflexion |
Neurodynamic Testing
PositiveĀ when the patientās familiar leg symptomsĀ are reproduced (not merely tightness).
Straight Leg Raise (SLR)
Supine; lift straight leg (ankle neutral).
Positive: symptom reproduction ~30ā70° hip flexion.
BragardāsĀ (add ankle dorsiflexion) can increase neural load/differentiate.
Slump Test
Sitting; thoracolumbar flexion + cervical flexion; extend knee & dorsiflex ankle.
Repeat asymptomatic ā symptomatic side.
Positive: reproduction of concordant symptoms during sequence.
Femoral Nerve Stretch Test
Prone (or sidelying); passively flex knee (and add hip extension).
Positive: anterior thigh/groin symptomsāuseful for L2āL4Ā involvement.
Repeated Movement Testing (MDT/McKenzie)
Goal: determine if leg pain centralizesĀ (moves proximally toward the back), indicating mechanical responsiveness.
Key considerations
Test multiple directionsĀ (not just extension): flexion, lateral shift, lateral flexion.
Assess in loadedĀ (standing) and unloadedĀ (lying) positions.
Use sufficient volume: often ā„10ā15 repetitions are needed for change.
Track: intensity, distributionĀ (centralization/peripheralization), ROM, posture.
Centralization is a favorable prognostic sign and may directly inform treatment direction.
Clinical Notes for Practice
Document baseline neuro statusĀ (myotomes, dermatomes, reflexes) to enable testāretestĀ after interventions.
Combine history clustersĀ (e.g., leg pain > back pain, dermatomal numbness, myotomal weakness, positive SLR/Slump) with exam findings for better accuracy.
Consider differential diagnoses (hip pathology, peripheral neuropathy, vascular claudication, referred pain from SIJ/hip) when the pattern is atypical.
Use imagingĀ judiciouslyāclinical course and response to treatment often guide need.
Sources:
Rainey N. Lumbar Radiculopathy Assessment Course. Plus, 2023.
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